![]() Epilepsy may mimic TGA in a minority of cases. The significant positive association between migraine and TGA may support this hypothesis. Since half of our patients had a precipitating event in their history, it is reasonable to hypothesize that spreading depression may play a role in TGA. The results of our case-control and longitudinal studies point to the conclusion that TGA and TIA do not share the same etiology. In three patients (4.5%), the TGA was eventually considered to be of epileptic origin. Migraine was more common in TGA patients than in both normal and TIA control subjects. Transient global amnesia is an alarming, sudden syndrome of reversible anterograde and shrinking retrograde memory dysfunction, lasting several hours and occurring in otherwise healthy adults. In contrast to TIA control subjects, no TGA patient suffered stroke, myocardial infarction, or TIA during the follow-up period. ![]() Clear-cut anterograde amnesia must be present during the attack. Ischemic heart disease, smoking, and peripheral vascular disease were associated with a significantly reduced risk for TGA compared with TIA. We did not find evidence of an increased risk of TGA associated with any vascular risk factor. Attacks must be witnessed and information must be available from a capable observer. Then we compared the outcome of the two groups using actuarial analysis based on survival curves. Various mechanisms have been proposed, including temporary metabolic abnormality in the medial temporal. We prospectively studied the vascular events and mortality rates of the TGA cases and of the TIA control subjects. The etiology of transient global amnesia is unclear. In a case-control study, we compared the prevalence of vascular risk factors in 64 TGA patients with 64 first-ever transient ischemic attack (TIA) control subjects and 108 normal community-based control subjects matched for age and sex. Cerebral venous hypertension due to incompetence of the internal jugular valve may play a role in the pathogenesis of TGA.The purpose of the present study was to make an attempt to ascertain the etiology of transient global amnesia (TGA), which is still disputed more than 30 years after the first description of this clinical entity. Cerebral venous hypertension due to incompetence of the internal jugular valve may play a role in the pathogenesis of TGA. A person having an attack of TGA has almost no. ![]() Intracerebral hemorrhages usually develop during minutes and. Other disorders that have acute onset and cause persistent focal signs should be considered in the differential diagnosis. The aim of this study was to identify potential risk factors for TGA, vascular risk factors, the role of patent foramen ovale (PFO. Ischemic strokes are characterized by the abrupt or at least very acute onset of focal neurologic symptoms and signs that leave persistent neurologic deficits. Paradoxical embolism due to PFO as a cause of TGA is not confirmed in our study. A patient with anterograde amnesia may not be able to form new memories indefinitely. The aetiology of transient global amnesia (TGA) is still unknown. Paradoxical embolism due to PFO as a cause of TGA is not confirmed in our study. Conclusions: TGA patients have fewer vascular risk factors than TIA patients. What causes transient global amnesia is not known, but a similar, temporary loss of memory can result from drinking too much alcohol or taking certain drugs. TGA patients have fewer vascular risk factors than TIA patients. Transient global amnesia is a sudden, temporary loss of memory for events during, after, and sometimes before the event that caused the amnesia. ACUV detected jugular valve incompetence in 72.9% TGA, 35.7% TIA and 39.5% controls (TGA vs. No statistical difference was found between the 3 groups with regard to PFO. TGA patients and controls showed a lower prevalence for vascular risk factors than TIA patients. ![]() Retrograde jugular venous flow was tested with air contrast ultrasound venography (ACUV). PFO was studied by contrast transcranial duplex sonography. The aim of this study was to identify potential risk factors for TGA, vascular risk factors, the role of patent foramen ovale (PFO) and of retrograde jugular venous flow.ฤก38 subjects entered the study, including 48 patients with TGA, 42 age-matched patients with transient ischaemic attack (TIA) and 48 controls. We attempted to determine risk factors for TGA, as well as prognostic factors that may cause recurrence. The aetiology of transient global amnesia (TGA) is still unknown. Aetiology of transient global amnesia (TGA) remains uncertain, though many have been proposed, including ischaemic, migrainous or epileptic pathologies. ![]()
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